Chapter 8 - Reflex evaluation
Questions
Define the following terms:
hyper-reflexia,
pathological spread of reflex,
clonus,
Babinski
sign,
Hoffmann's sign,
myotatic reflex,
upper
motor neurons,
lower motor neurons,
reinforcement.
Hyper-reflexia is excessively brisk reflexes
Pathological spread of reflex occurs when a reflex
contraction occurs in a muscle whose tendon was not stretched (i.e., finger
flexion when testing the brachioradialis reflex or thigh adduction when the
patellar reflex is tested). It is a suggestion of hyperactive reflexes.
Clonus is repeated contraction of muscles (usually the calf
muscles or the wrist flexor muscles) when the muscles are stretched manually
(such as by ankle dorsiflexion or wrist extension). Sustained clonus is when
this occurs repeatedly as long as the stretch is maintained.
Babinski sign is reflex dorsiflexion of the great toes and
fanning of the other toes by stroking the lateral side of the sole of the foot.
This stroke is often continued across the ball of the foot toward the base of
the great toe. This occurs in patients with upper motor neuron damage. The
normal plantar response is for the great toe to flex.
Hoffmann's sign is flexion of the thumb following a maneuver
that consists first of passive flexion of the patient's middle finger by
pressure over the nail bed, followed by sudden release of this pressure. It is
a sign of brisk reflexes but is not pathological unless it is accompanied by
other signs of upper motor neuron damage or is asymmetrical.
Myotatic reflex is the muscle stretch reflex (often termed
the deep tendon reflex).
Upper motor neurons are the principal descending motor
pathways for voluntary movement, including the corticospinal and corticobulbar
tracts (and some other associated tracts).
Lower motor neurons are the anterior horn motor neurons and
their axons that extend through the ventral nerve root and the peripheral
nerves to reach the neuromuscular junction.
Reinforcement involves the strong contraction of muscles outside of the area in which muscle stretch reflexes are being tested. This will serve to increase the reflexes. Specific examples include clenching the jaw, pressing the feet together or clasping the hands and attempting to pull them apart (the Jendrasik maneuver).
8-1. What is the
main effect of descending motor systems on reflexes?
Answer 8-1. Motor cortex and descending motor pathways are generally involved in suppressing
(inhibiting) reflexes.
8-2. What are the
7 Deep Tendon Reflex exams (DTRs)? What sensory/motor nerves are they testing?
Answer 8-2. Biceps - musculocutaneous nerve and mainly C6; Triceps - radial nerve and mainly C7; Brachioradialis (radial periosteal) - radial nerve and
mainly C6; Finger flexor - musculocutaneous nerve and mainly C7-8; Patellar - femoral nerve and mainly L3-L4; Achilles' reflex (ankle jerk) - tibial nerve and
mainly S1; Jaw jerk - trigeminal
8-3. What are the
superficial reflexes?
Answer 8-3. Superficial reflexes include: abdominal, cremaster,
plantar, anal wink.
8-4. What is the
effect of damage to corticospinal fibers on myotatic (deep tendon) reflexes?
What is the effect on superficial reflexes?
Answer 8-4. DTRs increase with damage to descending motor pathways;
superficial reflexes decrease with damage to descending motor pathways.
8-5. What primitive
reflexes emerge with diffuse bilateral hemispheric dysfunction?
Answer 8-5. Diffuse bilateral hemispheric dysfunction can
dysinhibit grasp, glabellar, suck, rooting, oculocephalic and nuchocephalic
reflexes.
8-6. What happens
to DTRs with lesions in the cerebellum & basal ganglia?
Answer 8-6. Usually no change, though may be sluggish with
cerebellar damage.
8-7. How are DTRs
graded?
Answer 8-7. 0-4+. To grade a reflex as "0", you must try reinforcement. 4+ means there is sustained
clonus. 1 is sluggish, 2 is "normal" and 3 is "brisk".
8-8. What is the
most important consideration in testing reflexes?
Answer 8-8. Symmetry.
8-9. What reflex
changes would occur in lesions of muscles?
Answer 8-9. No change
unless end stage.
8-10. What reflex
changes would occur in lesions of the neuromuscular junction?
Answer 8-10. Normal to decreased depending on severity of weakness.
8-11. What reflex
changes would occur in lesions of the peripheral nerves?
Answer 8-11. Decreased in clinically affected areas.
8-12. What reflex
changes would occur in lesions of the nerve root?
Answer 8-12. Decreased in clinically affected areas.
8-13. What reflex
changes would occur in lesions of the spinal cord and brain stem?
Answer 8-13. Usually reflexes will be increased unless the gray
matter (anterior horn cells, lower motor neurons) is damaged right at the
reflex level. Acute spinal cord injury can result in spinal cord shock
(flaccid, decreased reflex).
8-14. How can damage
to sensory nerve fibers affect reflexes?
Answer 8-14. Damage to sensory nerve fibers may also decrease
reflexes by damaging the afferent limb of the reflex arc.
8-15. What is the
effect of neuropathy on muscle stretch reflexes?
Answer 8-15. Neuropathy often produces decreased reflexes out of
proportion to weakness.
8-16. What are some
visceral reflexes that can be tested?
Answer 8-16. Visceral reflexes include: pupillary light reflex,
oculocardiac, carotid sinus, bulbocavernosus, rectal (internal sphincter) and
orthostatic blood pressure regulation.